Life Cycle of Antheraea mylitta

Study of Morphology, Life Cycle, Prevalence, Epidemiology, Pathogenicity, Diagnosis, Prophylaxis, and Treatment of Schistosoma haematobium

 

Introduction

Schistosoma haematobium is a parasitic trematode responsible for urinary schistosomiasis, a significant public health problem in endemic regions, primarily in Africa and the Middle East. This parasite inhabits the venous plexus of the urinary bladder and is transmitted through freshwater snails of the genus Bulinus.

Morphology

Schistosoma haematobium is a dioecious trematode with distinct male and female forms. The male is shorter and thicker (10–15 mm in length) with a gynecophoric canal to hold the female. The female is longer and slender (20 mm in length), residing within the male’s gynecophoric canal. Eggs are oval with a characteristic terminal spine, measuring about 112–170 µm in length.

Life Cycle

The life cycle of Schistosoma haematobium involves two hosts: a freshwater snail (Bulinus species) as the intermediate host and humans as the definitive host. The detailed life cycle is as follows:

1.      Eggs Excretion and Hatching:

The adult worms reside in the venous plexus of the urinary bladder, where the female lays eggs. These eggs travel through the bladder walls and are excreted in urine.Upon contact with freshwater, the eggs hatch into free-swimming miracidia within a few hours.

2.      Infection of Snail Host:

The miracidia actively search for and penetrate the soft tissues of the intermediate host, freshwater snails of the genus Bulinus. Inside the snail, they undergo asexual reproduction, developing into primary sporocysts and subsequently into secondary sporocysts. The sporocysts further mature and produce free-swimming cercariae, which are released into the water.

3.      Cercarial Penetration of Human Skin:

The cercariae are highly motile and actively seek human hosts when they come in contact with infested water. Using enzymes, the cercariae penetrate the skin, shedding their tails to become schistosomula.

4.      Migration and Maturation in Humans:

The schistosomula enter the bloodstream and migrate through the venous system to the lungs and liver. After maturing in the liver for about 4-6 weeks, they migrate to the venous plexus of the urinary bladder, where they mature into adult male and female worms.

5.      Egg Production and Pathogenesis:

The paired adult worms produce eggs, some of which are excreted through urine, while others remain trapped in tissues, causing inflammation and fibrosis. The cycle continues when the excreted eggs reach freshwater and hatch into miracidia.

Prevalence and Epidemiology

1.       Found predominantly in Africa, the Middle East, and parts of the Indian Ocean islands.

2.       Transmission is highest in regions with stagnant freshwater bodies, poor sanitation, and human water contact.

3.       Children and agricultural workers are at higher risk.

Pathogenicity

1.       The disease primarily affects the urinary bladder, causing hematuria (bloody urine).

2.       Chronic infection leads to fibrosis, calcification, and bladder dysfunction.

3.       Long-term complications include squamous cell carcinoma of the bladder and kidney damage.

4.       Egg deposition in tissues induces granuloma formation and inflammation.

Diagnosis

1.       Microscopic Examination: Detection of eggs with a terminal spine in urine samples.

2.       Serological Tests: ELISA and PCR for antigen/antibody detection.

3.       Imaging Techniques: Ultrasound to assess bladder and kidney damage.

4.       Urinalysis: Hematuria and proteinuria indicate infection.

Prophylaxis

1.       Avoiding contact with contaminated water.

2.       Proper disposal of human waste to prevent snail contamination.

3.       Snail control measures (molluscicides, environmental management).

4.       Health education and awareness campaigns.

Treatment

1.       Praziquantel (40 mg/kg body weight, single dose) is the drug of choice.

2.       Repeated treatment may be required in highly endemic areas.

3.       Supportive therapy for complications like bladder cancer and kidney failure.

 

References

1.      Gryseels, B., Polman, K., Clerinx, J., & Kestens, L. (2006). Human schistosomiasis. The Lancet, 368(9541), 1106-1118.

2.      Colley, D. G., Bustinduy, A. L., Secor, W. E., & King, C. H. (2014). Human schistosomiasis. The Lancet, 383(9936), 2253-2264.

3.      WHO (2023). Schistosomiasis. World Health Organization Website. https://www.who.int

4.      Centers for Disease Control and Prevention (CDC). (2023). Schistosomiasis. CDC Parasitic Diseases Website. https://www.cdc.gov

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